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E-Only Articles
An in vitro model of warm hypoxia–reoxygenation injury in human liver endothelial cells
Ischemia–reperfusion or hypoxia–reoxygenation (H-R) injury adversely affects hepatic function following transplantation and major resection; the death of human sinusoidal endothelial cells (SECs) by apoptosis may play a central role in this process. Caspase-3 is an important intracellular protease in the intrinsic and extrinsic pathways of apoptosis.
